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dc.contributor.advisorWhaley-Connell, Adam T.eng
dc.contributor.authorPanfili, Zacharyeng
dc.contributor.authorHabibi, Javad, 1947-eng
dc.contributor.authorHayden, Melvineng
dc.contributor.authorNistala, Ravieng
dc.contributor.authorParrish, Alaneng
dc.contributor.authorSowers, James R. (James Russell), 1942-eng
dc.contributor.authorTilmon, Rogereng
dc.contributor.corporatenameUniversity of Missouri--Columbia. School of Medicineeng
dc.date.issued2010eng
dc.description.abstractActivation of the renin-angiotensin system (RAS) and subsequent elevations of tissue angiotensin (Ang) II contribute to the development of proteinuria and progressive kidney disease. Recent work suggests that proximal tubule injury contributes to development of proteinuria in addition to glomerular contributions. Thereby, the aim of this study was to determine the impact that Ang II has on renal proximal tubular cell (PTC) function in a transgenic rodent model of hypertension and nephropathy, the transgenic TG(mRen2)27 rat.eng
dc.identifier.urihttp://hdl.handle.net/10355/9158
dc.languageEnglisheng
dc.relation.ispartofcommunityUniversity of Missouri--Columbia. Health Sciences Research Day.eng
dc.subjectkidney damageeng
dc.subjecttubolinterstitial fibrosiseng
dc.subject.FASTRenin-angiotensin systemeng
dc.subject.FASTKidneys -- Diseaseseng
dc.titleAng II contributes proximal tubule remodeling in transgenic Ren2 rats [abstract]eng
dc.typeOthereng


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