dc.contributor.author | Waggoner, Michael | eng |
dc.contributor.author | Tandon, Ashish | eng |
dc.contributor.author | Tovey, Jonathan | eng |
dc.contributor.author | Sharma, Ajay | eng |
dc.contributor.author | Lopez, Vanessa | eng |
dc.contributor.author | Cowden, John W. | eng |
dc.contributor.author | Rieger, Frank G., III | eng |
dc.contributor.author | Hamm, Chuck W. | eng |
dc.contributor.meetingname | Health Sciences Research Day (2010 : University of Missouri) | eng |
dc.date.issued | 2010 | eng |
dc.description.abstract | TGF[beta] induces the transformation of corneal keratocytes into fibroblasts and myofibroblasts resulting in the formation of corneal haze (scar) following injury. We investigated whether epigenetic modifications can prevent development of corneal haze in vivo using a rabbit model. | eng |
dc.format.extent | 1 page | eng |
dc.identifier.uri | http://hdl.handle.net/10355/9407 | |
dc.language | English | eng |
dc.relation.ispartofcommunity | University of Missouri--Columbia. Health Sciences Research Day | eng |
dc.rights | OpenAccess. | eng |
dc.rights.license | This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 3.0 License. | eng |
dc.subject | eye injury | eng |
dc.subject | occluded vision | eng |
dc.title | SAHA : FDA approved histone deacetylase inhibitor demonstrates exceptionally high inhibition of corneal haze following PRK surgery in rabbit model [abstract] | eng |
dc.type | Abstract | eng |