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dc.contributor.authorRichter, Catherine A.eng
dc.contributor.authorTaylor, Julia A.eng
dc.contributor.authorRuhlen, Rachel L.eng
dc.contributor.authorvom Saal, Frederick S.eng
dc.date.issued2007-06eng
dc.descriptiondoi:10.1289/ehp.9804eng
dc.description.abstractHormonal alterations during development have lifelong effects on the prostate gland. Endogenous estrogens, including 17β-estradiol (E2), and synthetic estrogenic endocrine disruptors, such as bisphenol A (BPA), have similar effects on prostate development. Increasing exposure to estrogens within the low-dose, physiologic range results in permanent increases in the size and androgen responsiveness of the prostate, whereas exposure within the high-dose, pharmacologic range has the opposite effects.eng
dc.identifier.citationEnvironmental Health Perspectives, 115(6), 2007.eng
dc.identifier.urihttp://hdl.handle.net/10355/8675eng
dc.publisherEnvironmental Health Perspectiveseng
dc.relation.ispartofBiological Sciences publications (MU)eng
dc.relation.ispartofcommunityUniversity of Missouri-Columbia. College of Arts and Sciences. Division of Biological Scienceseng
dc.subjectandrogen receptor geneeng
dc.subjectsexual differentiationeng
dc.subjectdose-response relationshipeng
dc.subjectestrogen receptor 1eng
dc.subject.lcshMice -- Development -- Endocrine aspectseng
dc.subject.lcshHormones -- Receptorseng
dc.subject.lcshProstateeng
dc.titleEstradiol and Bisphenol A Stimulate Androgen Receptor and Estrogen Receptor Gene Expression in Fetal Mouse Prostate Mesenchyme Cellseng
dc.typeArticleeng


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