dc.contributor.author | Richter, Catherine A. | eng |
dc.contributor.author | Taylor, Julia A. | eng |
dc.contributor.author | Ruhlen, Rachel L. | eng |
dc.contributor.author | vom Saal, Frederick S. | eng |
dc.date.issued | 2007 | eng |
dc.description | doi:10.1289/ehp.9804 | eng |
dc.description.abstract | Hormonal alterations during development have lifelong effects on the prostate gland. Endogenous estrogens, including 17β-estradiol (E2), and synthetic estrogenic endocrine disruptors, such as bisphenol A (BPA), have similar effects on prostate development. Increasing exposure to estrogens within the low-dose, physiologic range results in permanent increases in the size and androgen responsiveness of the prostate, whereas exposure within the high-dose, pharmacologic range has the opposite effects. | eng |
dc.identifier.citation | Environmental Health Perspectives, 115(6), 2007. | eng |
dc.identifier.uri | http://hdl.handle.net/10355/8675 | eng |
dc.publisher | Environmental Health Perspectives | eng |
dc.relation.ispartof | Biological Sciences publications (MU) | eng |
dc.relation.ispartofcommunity | University of Missouri-Columbia. College of Arts and Sciences. Division of Biological Sciences | eng |
dc.subject | androgen receptor gene | eng |
dc.subject | sexual differentiation | eng |
dc.subject | dose-response relationship | eng |
dc.subject | estrogen receptor 1 | eng |
dc.subject.lcsh | Mice -- Development -- Endocrine aspects | eng |
dc.subject.lcsh | Hormones -- Receptors | eng |
dc.subject.lcsh | Prostate | eng |
dc.title | Estradiol and Bisphenol A Stimulate Androgen Receptor and Estrogen Receptor Gene Expression in Fetal Mouse Prostate Mesenchyme Cells | eng |
dc.type | Article | eng |